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Aging, Reproduction, and the Climacteric by Caleb E. Finch, Roger G. Gosden (auth.), Luigi Mastroianni

By Caleb E. Finch, Roger G. Gosden (auth.), Luigi Mastroianni Jr., C. Alvin Paulsen (eds.)

Interest in sexuality and reproductive functionality doesn't stop whilst humans start to age. as a substitute, a brand new set of questions arises. girls need to know whether it is secure to have infants of their overdue thirties and early forties. they need to grasp extra approximately sizzling flashes and different indicators of menopause-which ones are harmful and that are in simple terms uncomfortable. they're wanting to know about the relative hazards and merits of estrogen alternative remedy. males, too, are thinking about age-related alterations of their sexual functionality. specialists in reproductive body structure, gerontology, and genetics met on the nationwide Institutes of wellbeing and fitness in June of 1984 to debate those and different matters approximately getting older and the reproductive procedure. The convention on getting older, Reproduc­ tion, and the Climacteric used to be backed through the yankee Fertility Society, The nationwide Institute on getting older, and the nationwide Institute of kid future health and Human improvement. This quantity relies at the lawsuits of that confer­ ence.

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And Klima, W. , 1978, Diseases of untreated virgin female REM and BALB/c mice, J. Gerontol. 33:178-183. , and Mahesh, V. , 1975, Primordial follicles with nonnal oocytes in the ovaries of postmenopausal women, J. Am. Geriatr. Soc. 23:193-196. Court Brown, W. , and Smith, P. , 1969, Sex chromosome aneuploidy and parental age, Ann. Hum. Gent. 33:1-14. Cramer, D. , Welch, W. , and Scully, R. , 1983, Mumps, menarche, menopause, and ovarian cancer, Am. J. Obstet. Gynecol. 147:1-6. Crowder, M. , and Nett, T.

And Russell, B. , 1957, Gene-induced embryological modifications of primordial germ cells in the mouse, 1. Exp. Zool. 134:207-237. Mobbs, C. , Flurkey, K. , Gee, D. , Sinha, Y. , and Finch C. , 1984a, Estradiol-induced anovulatory syndrome in female C57BLl6J mice: Age-like neuroendocrine, but not ovarian impairments, Bioi. Reprod. 30:556-563. Mobbs, C. , Gee, D. , and Finch, C. , 1984b, Reproduction in senescence in female C57BLl6J mice: Ovarian impairments and neuroendocrine impairments that are partially reversible and delay able by ovariectomy, Endocrinology 116:813-820.

1979; Roth and Hess, 1982), but it is unclear whether the loss is due to a decrease of E2 binding sites per cell, or to a loss of E2 -binding cells. Osteoporosis The view prevailing since the classic study of Albright, et al. (1941) holds that deficiences in estrogens are a major cause of postmenopausal osteoporosis. , 1983). , 1967; Avioli, 1982). , 1983), which is 5-10 years before significant decreases in circulating E2 . Thus, osteoporosis in women is not precipitated only by sex steroid loss at menopause.

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